cox proportional hazards regression model Search Results


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Renin Phenotype and the <t> multivariable </t> adjusted risk for incident hypertension.
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( A ) Immunohistochemistry staining for LGALS3BP, S100A8/A9, CHI3L1, and MMP9 plus SMC (SMA, CNN1) and macrophage (CD68) markers. Scale bars: 500 μm. ( B ) Association between the biomarker candidates and atherosclerosis ( n = 560) and CVD ( n = 685) in the Bruneck study. Direction and strength of associations are coded by color and significance levels by size of frames. ORs are derived from logistic regression analysis, and <t>hazard</t> ratios from <t>Cox</t> <t>models.</t> ORs and hazard ratios are expressed for a 1-SD higher log e -transformed level of the given marker. Full details are presented in . Model 1 (M1) was adjusted for age (years) and sex. M2 was additionally controlled for standard vascular risk factors, including LDL cholesterol (mg/dl), HDL cholesterol (mg/dl), high-sensitivity CRP (mg/l), diabetes mellitus (0 vs. 1), hypertension (0 vs. 1), smoking (pack-years), and body mass index (kg/m 2 ). *Additionally adjusted for atherosclerosis score (mm).
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Image Search Results


Renin Phenotype and the multivariable adjusted risk for incident hypertension.

Journal: Annals of internal medicine

Article Title: The Spectrum of Subclinical Primary Aldosteronism and Incident Hypertension: A Cohort Study

doi: 10.7326/M17-0882

Figure Lengend Snippet: Renin Phenotype and the multivariable adjusted risk for incident hypertension.

Article Snippet: We used multivariable discrete Cox proportional hazards models (proc PHREG, ties=discrete, SAS v9.4) to evaluate the association between serum aldosterone and incident hypertension by renin phenotypes and report the results as hazard ratios( 29 ).

Techniques: Clinical Proteomics, Activity Assay

Aldosterone (continuous) and the multivariable adjusted relative risk for incident hypertension by renin phenotype.

Journal: Annals of internal medicine

Article Title: The Spectrum of Subclinical Primary Aldosteronism and Incident Hypertension: A Cohort Study

doi: 10.7326/M17-0882

Figure Lengend Snippet: Aldosterone (continuous) and the multivariable adjusted relative risk for incident hypertension by renin phenotype.

Article Snippet: We used multivariable discrete Cox proportional hazards models (proc PHREG, ties=discrete, SAS v9.4) to evaluate the association between serum aldosterone and incident hypertension by renin phenotypes and report the results as hazard ratios( 29 ).

Techniques: Clinical Proteomics, Activity Assay

( A ) Immunohistochemistry staining for LGALS3BP, S100A8/A9, CHI3L1, and MMP9 plus SMC (SMA, CNN1) and macrophage (CD68) markers. Scale bars: 500 μm. ( B ) Association between the biomarker candidates and atherosclerosis ( n = 560) and CVD ( n = 685) in the Bruneck study. Direction and strength of associations are coded by color and significance levels by size of frames. ORs are derived from logistic regression analysis, and hazard ratios from Cox models. ORs and hazard ratios are expressed for a 1-SD higher log e -transformed level of the given marker. Full details are presented in . Model 1 (M1) was adjusted for age (years) and sex. M2 was additionally controlled for standard vascular risk factors, including LDL cholesterol (mg/dl), HDL cholesterol (mg/dl), high-sensitivity CRP (mg/l), diabetes mellitus (0 vs. 1), hypertension (0 vs. 1), smoking (pack-years), and body mass index (kg/m 2 ). *Additionally adjusted for atherosclerosis score (mm).

Journal: The Journal of Clinical Investigation

Article Title: Extracellular matrix proteomics identifies molecular signature of symptomatic carotid plaques

doi: 10.1172/JCI86924

Figure Lengend Snippet: ( A ) Immunohistochemistry staining for LGALS3BP, S100A8/A9, CHI3L1, and MMP9 plus SMC (SMA, CNN1) and macrophage (CD68) markers. Scale bars: 500 μm. ( B ) Association between the biomarker candidates and atherosclerosis ( n = 560) and CVD ( n = 685) in the Bruneck study. Direction and strength of associations are coded by color and significance levels by size of frames. ORs are derived from logistic regression analysis, and hazard ratios from Cox models. ORs and hazard ratios are expressed for a 1-SD higher log e -transformed level of the given marker. Full details are presented in . Model 1 (M1) was adjusted for age (years) and sex. M2 was additionally controlled for standard vascular risk factors, including LDL cholesterol (mg/dl), HDL cholesterol (mg/dl), high-sensitivity CRP (mg/l), diabetes mellitus (0 vs. 1), hypertension (0 vs. 1), smoking (pack-years), and body mass index (kg/m 2 ). *Additionally adjusted for atherosclerosis score (mm).

Article Snippet: We analyzed the case-cohort data from the SAPHIR study using Cox proportional hazard models with Prentice weights and robust standard errors ( ).

Techniques: Immunohistochemistry, Staining, Biomarker Discovery, Derivative Assay, Transformation Assay, Marker